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The cellular modifier MOAG‐4/SERF drives amyloid formation through charge  complementation | The EMBO Journal
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal

Probing the Origin of the Toxicity of Oligomeric Aggregates of α-Synuclein  with Antibodies | ACS Chemical Biology
Probing the Origin of the Toxicity of Oligomeric Aggregates of α-Synuclein with Antibodies | ACS Chemical Biology

Julius Bier Kirkegaard — Theoretical Biophysics & Data Science
Julius Bier Kirkegaard — Theoretical Biophysics & Data Science

Delivery of Native Proteins into C. elegans Using a Transduction Protocol  Based on Lipid Vesicles | Scientific Reports
Delivery of Native Proteins into C. elegans Using a Transduction Protocol Based on Lipid Vesicles | Scientific Reports

Frontiers | Regulation of Age-Related Protein Toxicity
Frontiers | Regulation of Age-Related Protein Toxicity

Crystal Structure and Mechanism of Tryptophan 2,3-Dioxygenase, a Heme  Enzyme Involved in Tryptophan Catabolism and in Quinolinate Biosynthesis, |  Biochemistry
Crystal Structure and Mechanism of Tryptophan 2,3-Dioxygenase, a Heme Enzyme Involved in Tryptophan Catabolism and in Quinolinate Biosynthesis, | Biochemistry

Speakers | ARDD 2022
Speakers | ARDD 2022

MOAG-4 promotes the aggregation of α-synuclein by competing with  self-protective electrostatic interactions - ScienceDirect
MOAG-4 promotes the aggregation of α-synuclein by competing with self-protective electrostatic interactions - ScienceDirect

Deletion of SERF2 in mice delays embryonic development and alters amyloid  deposit structure in the brain | Life Science Alliance
Deletion of SERF2 in mice delays embryonic development and alters amyloid deposit structure in the brain | Life Science Alliance

Proteome-wide Changes in Protein Turnover Rates in C. elegans Models of  Longevity and Age-Related Disease - ScienceDirect
Proteome-wide Changes in Protein Turnover Rates in C. elegans Models of Longevity and Age-Related Disease - ScienceDirect

The cellular modifier MOAG‐4/SERF drives amyloid formation through charge  complementation | The EMBO Journal
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal

Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity:  Cell
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity: Cell

Cognitive impairments correlate with increased central nervous system  immune activation after allogeneic haematopoietic stem cell transplantation  | Leukemia
Cognitive impairments correlate with increased central nervous system immune activation after allogeneic haematopoietic stem cell transplantation | Leukemia

Protein quality control: From mechanisms to disease
Protein quality control: From mechanisms to disease

Frontiers | Key Metabolic Pathways in MSC-Mediated Immunomodulation:  Implications for the Prophylaxis and Treatment of Graft Versus Host Disease
Frontiers | Key Metabolic Pathways in MSC-Mediated Immunomodulation: Implications for the Prophylaxis and Treatment of Graft Versus Host Disease

The cellular modifier MOAG‐4/SERF drives amyloid formation through charge  complementation | The EMBO Journal
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal

Cornelis CALKHOVEN | University of Groningen, Groningen | RUG | European  Research Institute for the Biology of Ageing | Research profile
Cornelis CALKHOVEN | University of Groningen, Groningen | RUG | European Research Institute for the Biology of Ageing | Research profile

A Brain-Permeable Aminosterol Regulates Cell Membranes to Mitigate the  Toxicity of Diverse Pore-Forming Agents | ACS Chemical Neuroscience
A Brain-Permeable Aminosterol Regulates Cell Membranes to Mitigate the Toxicity of Diverse Pore-Forming Agents | ACS Chemical Neuroscience

MOAG-4 promotes the aggregation of α-synuclein by competing with  self-protective electrostatic interactions - ScienceDirect
MOAG-4 promotes the aggregation of α-synuclein by competing with self-protective electrostatic interactions - ScienceDirect

Polyglutamine protein aggregates are dynamic | Nature Cell Biology
Polyglutamine protein aggregates are dynamic | Nature Cell Biology

Speakers | ARDD 2022
Speakers | ARDD 2022

Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity:  Cell
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity: Cell

Tobacco carcinogen induces tryptophan metabolism and immune suppression via  induction of indoleamine 2,3-dioxygenase 1 | Signal Transduction and  Targeted Therapy
Tobacco carcinogen induces tryptophan metabolism and immune suppression via induction of indoleamine 2,3-dioxygenase 1 | Signal Transduction and Targeted Therapy

Probiotic Bacillus subtilis Protects against α-Synuclein Aggregation in C.  elegans - ScienceDirect
Probiotic Bacillus subtilis Protects against α-Synuclein Aggregation in C. elegans - ScienceDirect